ADALBERTO MOSQUEDA TAYLOR PDF

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Adalberto Mosqueda-Taylor’s 2 research works with 39 reads, including: Oct-4 and CD44 in epithelial stem cells like of benign odontogenic lesions. Adalberto. Adalberto Mosqueda of Metropolitan Autonomous University, Mexico City (UAM). Toussaint-Caire S, Vega-Memíje ME, Mosqueda-Taylor A, Cuevas-González. Adalberto Mosqueda Taylor is the author of Oral Medicine And Pathology At A Glance ( avg rating, 12 ratings, 0 reviews, published ), Oral Medicin.

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Adalberto Mosqueda Taylor – Böcker | Bokus bokhandel

Basu A, Haldar S. Odontogenic tumors in Mexico: Oral oncology 40 10, Angiocentric lymphomas of the palate: These facts led us to consider that this lesion mimics the early primordial stages of tooth development. The pathogenesis of oral MA remains uncertain, although its clinical Silicone granuloma of the facial tissues: Please review our privacy policy. When it does occur; its clinical features resemble those of sarcomatoid carcinoma, and its biologic behavior is similar to that of malignant mesenchymal neoplasms.

Odontogenic tumors: A retrospective study of four Brazilian diagnostic pathology centers

This protein, which is synthesized in young ameloblasts, is secreted into enamel matrix 10 and is gradually almost entirely removed by degradation by extracellular enzymes during enamel maturation There was not significant difference between both periods in terms of tumor size 6.

The pathogenesis of oral MA remains uncertain, although its clinical behavior is suggestive of a reactive origin.

On the other hand, the cytoplasmic and membranous expression also suggests the co-existence of different stages of maturation in this epithelium. The male-female ratio was 1.

Journal of Clinical and Experimental Dentistry.

Oral oncology 41 8, Glucose transporter type1 GLUT-1 deficiency. To discuss the convenience of laser surgery as optimal treatment for melanoma of the oral mucosa.

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Age ranged from 25 to 72 years mean: Several reports on series of OT from different countries have documented distinct geographic variations in tumor prevalence, with the main difference being the relative incidence of ameloblastoma and odontoma 4581016 Data from the literature show differences in the relative frequencies of these tumors 3 – There were 14 female and 6 male patients.

The first internationally accepted classification system for these tumors was published in by the World Health Organization WHOwhich was reviewed and updated in and in 2. Although several CKs are expressed in the epithelial cells of the tooth germ 6CK 14 is the one that seems to be present constantly in all stages of tooth development and in most adwlberto epithelial cells adalbrto odontogenic tumors 7 – 9in contrast to CK 19, which increases its expression once the inner epithelial cells become potential ameloblasts 8.

Adalberto Abel Mosqueda Taylor –

Therefore, as has been pointed out in previous studies, the relatively moequeda frequency of malignant tumors observed in this and some other studies may be a reflection of the types of pathology services surveyed 5 Discussion This gaylor widely describes the immunohistochemical profile of POT. Extent of margins in soft tissue is the limiting factor for local control.

This observation is also reinforced by the fact that some qdalberto of the tumor epithelium showed focal positivity to vimentin Fig. The distribution of histological types and the relative frequencies of OT by centers of diagnosis are shown in Table 2.

Considering and putting together all these immunohistochemical findings, we interpret that the lesion studied is an odontogenic tumor of benign behavior that possibly originates from the mesenchyme of an abortive tooth germ that fails to produce a dental organ.

In addition, it has been reported that the pattern of Syndecan-1 expression varies according to the stage of maturation of the epithelial tissues Reduction in membranous immunohistochemical staining for the intracellular domain of epithelial cell adhesion molecule correlates with poor patient outcome in primary colorectal adenocarcinoma.

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Pathology international 59 4, Odontogenic tumors OT comprise an infrequent group of lesions arising from the tooth-producing tissues or its remnants, and asalberto range from hamartomatous or non-neoplastic proliferations to benign and malignant neoplasm with variable aggressiveness and metastatic potential 12.

Descriptive statistics were used in a preliminary analysis of the relationship between baseline variables. Expression of the CD34 gene in vascular endothelial cells.

The standardization of the examiners showed a kappa index of 0. As a member of the family of transmembrane heparan sulfate proteoglycans, Syndecan-1 CD is involved in cell to cell adhesion and in the interaction of cells with the extracellular matrix, and also participates in promoting cell proliferation and regulates cellular growth by interacting with families of growth factors in adalberrto with heparin The average age of presentation is Additionally, it is important to highlight the fact that there was a significantly higher proliferative index among the cells located within the subepithelial mesenchymal condensation as compared to that found in other mesenchymal areas of the tumor Fig.

The same group found that Syndecan-1 was intensely expressed in the mesenchyme, but not in the epithelium, by proliferating cells during the process of odontogenesis, suggesting a possible interaction of these tissues with growth factors Fig. Calcifying epithelial odontogenic tumor-like areas are common findings in adenomatoid odontogenic tumors and not a specific entity more.

There were six odontogenic carcinomas two malignant ameloblastomas, two clear cell odontogenic carcinomas, one primary intraosseous carcinoma and one carcinoma arising in an odontogenic cyst and one ameloblastic fibrosarcoma.